Cancer Drugs Used To Shrink Tumors In Fact Promote Their Growth
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In conventional cancer treatment, the focus is almost totally on the tumor – killing it via burning (radiation) or poisoning (chemotherapy), or removing it using surgery. Cancer drugs are also used to attempt to shrink tumors. Little, if anything, is usually done to improve the nutritional status, overall health, immunity and outlook of the person.
This is because conventional medicine views the tumor as the disease. If I’m not wrong, the word “oncology” actually means the study of tumors. Natural health and healing, on the other hand, is much wiser, viewing cancer as a systemic, whole-body disease. Any treatment then tackles the person as a whole.
With conventional medicine’s flawed understanding of the disease, also comes its highly limited and ineffective treatment of cancer. How well does conventional medicine work?
Recent research at the Moores Cancer Center at the University of California, San Diego has revealed that certain cancer drugs which are supposed to help shrink tumors in fact promote their growth! This is quite frightening, especially considering that thousands of cancer sufferers out there are probably being put on these drugs as we speak.
Read on for an article on the subject by Sherry Baker.
Cancer Drugs Make Tumors Grow
by Sherry Baker
(NaturalNews) Drugs like Avastin that are used to treat some cancers are supposed to work by blocking a vessel growth-promoting protein called vascular endothelial growth factor, or VEGF. With VEGF held in check, researchers have assumed tumors wouldn’t generate blood vessels and that should keep malignancies from growing. In a sense, the cancerous growths would be “starved”. But new research just published in the journal Nature shows this isn’t true. Instead of weakening blood vessels so they won’t “feed” malignant tumors, these cancer treatments, known as anti-angiogenesis drugs, actually normalize and strengthen blood vessels — and that means they can spur tumors to grow larger.
For their study, researchers at the Moores Cancer Center at the University of California, San Diego (UCSD) in La Jolla, replicated the action of anti-angiogenesis drugs by genetically decreasing VEGF levels in mouse tumors and inflammatory cells in several types of cancers, including pancreatic cancer. The research team, headed by David Cheresh, Ph.D., professor and vice chair of pathology at UCSD, also used drugs to inhibit VEGF receptor activity. The results? In every single instance, blood vessels were not weakened but, instead, were made normal again. And in some cases, tumors increased in size..
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